DON'T BE ALCOHOLIC

Drink No Liquor Johnny Depp caricature peace be still in nomine Patris et FiLii et Spiritus Sancti amen

Don't be an alcoholic it is written in the Holy King James Bible the word of the Lord according to King Solomon 950 BC Before Christ Proverbs 20:1 Wine is a mocker, strong drink is raging: & whosoever is deceived thereby is not wise. amen Common causes of low magnesium include: Alcohol use. Burns that affect a large area of the body. Chronic diarrhea. in nomine Patris et FiLii et Spiritus Sancti peace be still
https://www.youtube.com/watch?v=ahyPwQy6WcM
Six Reasons I Don't Drink Alcohol - Pastor Steve Gaines
https://www.youtube.com/watch?v=h8UsM1U6bKg
Intervention: Addicted to Alcohol - Most Viewed Moments | A&E

Don't drink beer !! it is written in the Holy King James Bible epistle to the Ephesians of saint Paul the apostle 50 AD Anno Domini in the year of our Lord Jesus Christ Ephesians 5:18 & be not drunk with wine, wherein is excess; but be filled with the Spirit; amen featuring a beer belly Obama sitting drinking beer Canadian flag to the right of Obama American flag to the left of Obama barn in the back one 6 pack beer to the side of the American flag Heineken 4pack bellow the Canadian flag enjoy this funny poster it is written in the Holy King James Bible the word of the Lord by isaiah the prophet of israel 600 BC Before Christ Isaiah 28:3 The crown of pride, the drunkards of Ephraim, shall be trodden under feet: amen Consumption of Specific Foods. Telomere length is positively associated with the consumption of Phosphorus, Hydrogen , Oxygen, Nitrogen & Carbon mixed with minerals whereas telomere length is inversely associated with consumption of alcohol, red meat, or processed meat , microglia of alcoholics showed elevated expression of genes involved in cell division and reduced expression of genes related to immune signaling and antigen presentation. After alcohol exposure, microglia from high-risk individuals exhibited more pronounced changes. Both high- and low-risk cells showed activation, but high-risk cells shifted more dramatically from a branched, resting state to a rounded, activated shape—indicative of heightened immune reactivity. These cells also displayed stronger gene expression changes, particularly in pathways related to immune responses and phagocytosis—the process by which microglia engulf and break down cellular debris, pathogens, or even synapses. One gene, CLEC7A, stood out. It was significantly upregulated in high-risk microglia following ethanol exposure. This gene encodes a receptor involved in recognizing foreign particles and initiating phagocytosis. Follow-up experiments confirmed that CLEC7A protein levels also rose, and phagocytic activity increased substantially in high-risk cells—both when using artificial particles and synaptic material from neurons “What stood out was the distinct difference in microglial behavior between individuals with high and low genetic risk for alcohol use disorder,” Pang told PsyPost. “The enhanced phagocytic activity and elevated CLEC7A expression in high-PRS microglia after ethanol exposure suggest that these cells are biologically primed to respond differently to alcohol, which we did not fully anticipate.” They created co-cultures combining neurons and microglia and observed the effects of alcohol. In the absence of microglia, neurons exposed to alcohol showed increased synaptic activity. This effect persisted when low-risk microglia were added. But when neurons were co-cultured with high-risk microglia, alcohol no longer enhanced synaptic connections—suggesting that these immune cells were pruning or removing synapses in response to ethanol. These results highlight a potential mechanism through which genetic predisposition could heighten vulnerability to alcohol use disorder. If microglia in genetically susceptible individuals are more prone to immune activation and synapse pruning, alcohol might accelerate the loss of healthy brain connectivity, potentially reinforcing problematic behaviors and impairing cognitive function.  “Genetic risk for alcohol use disorder doesn’t just influence behavior—it may also alter how the brain’s immune cells respond to alcohol,” Pang explained. “People with a higher genetic risk for alcohol use disorder may have brain immune cells (called microglia) that react more strongly to alcohol. This heightened response could lead to unnecessary trimming of brain connections over time, which may change how the brain functions and, ultimately, how a person behaves, such as increasing their risk of heavy drinking.” Common causes of low magnesium include: Alcohol use. Burns that affect a large area of the body. Chronic diarrhea. in nomine Patris et Filii et Spiritus Sancti amen
https://www.youtube.com/watch?v=Es8WmXghRYc
My Descent Into DARKNESS with ALCOHOL (Near-Death Experience)